Original articleIntrauterine growth retardation and blood pressure at age seven and eighteen
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Cited by (107)
Mechanical characterization of arteries affected by fetal growth restriction in guinea pigs (Cavia porcellus)
2018, Journal of the Mechanical Behavior of Biomedical MaterialsCitation Excerpt :The modeled pressurization retrieved an approximation of the potential behavior under a range of normal fetal arterial pressure (0–40 mmHg). Studies in healthy and restricted fetal sheep showed no differences in the mean pressure near term (Edwards et al., 1999; Louey et al., 2000), and also in human childhood and adolescence no significant differences have been established (Keijzer-Veen et al., 2005; Williams et al., 1992). Therefore, for the aims of this study, the blood pressure was assumed to be similar between healthy and restricted fetuses, using 28 ± 0.5 mmHg as reported for guinea pig fetuses (Kihlström, 1981).
Factors associated with growth patterns from birth to 18 months in a Beninese cohort of children
2014, Acta TropicaCitation Excerpt :Some authors have found that overall 60–80% of infants who die during the neonatal period are children born with LBW (Lawn et al., 2005). In addition, LBW is responsible for restricted growth in length and weight beginning in the neonatal period and continuing through childhood (ACC/SCN, 2000b), and this has been shown whether the mechanism is prematurity (Cooke et al., 2004; Gibson et al., 2006) or IUGR (Westwood et al., 1983; Williams et al., 1992). Moreover, IUGR and prematurity have been found associated with neonatal and perinatal morbidity (Froen et al., 2004; Ngoc et al., 2006; Richardus et al., 2003; Yasmin et al., 2001) and IUGR is also associated with a higher risk of appearance of cardiometabolic disorders later on in life (Barker et al., 1989; Gluckman et al., 2005).
Early childhood diarrhea and cardiometabolic risk factors in adulthood: The Institute of Nutrition of Central America and Panama Nutritional Supplementation Longitudinal Study
2013, Annals of EpidemiologyCitation Excerpt :The developmental origins of health and disease paradigm postulates that perturbations in the homeostasis of a developing fetus or child result in long-term changes affecting that individual's risk of future disease [1]. Individuals born at low birth weight [2–9] and those with low body mass index-for-age z-scores (BMIZ) in early childhood [10–12] have been found to be more likely to develop high blood pressure (BP), dyslipidemia, and insulin resistance—all features of the metabolic syndrome (MetS). Although the mechanisms behind these long-term outcomes are unknown, one plausible mechanism is through epigenetic changes in response to nutrient deficiency [13] and inflammatory signals [14].
Abnormal Circadian Blood Pressure Regulation in Children Born Preterm
2007, Journal of PediatricsRelationship between body height and dementia
2005, American Journal of Geriatric PsychiatryCitation Excerpt :An alternative and/or complementary explanation for our results is the association of growth hormone (GH) levels to both height and cognition. Cognitive impairment was related to GH deficiency in a study examining men suffering from isolated growth hormone deficiency.30,36. A study evaluating the effects of 2 years of GH therapy on cognitive performance in adults with childhood-onset growth hormone deficiency concluded that GH replacement therapy improved cognition in those subjects.37