Development of EEG and daytime sleep patterns in normal full-term infants during the first 3 months of life: Longitudinal observationsDéveloppement de l'EEG et des patterns de sommeil diurne chez des nourrissons à terme au cours des trois premiers mois de la vie: observations longitudinales☆
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Cited by (92)
The effect of napping and nighttime sleep on memory in infants
2021, Advances in Child Development and BehaviorCitation Excerpt :NREM Stage 3 is characterized by slow, synchronous, high voltage waves visible in the EEG and is therefore referred to as slow-wave-sleep (SWS). Sleep spindles are bursts of synchronized, oscillatory activity between 10 and 15 Hz which are present during NREM sleep and emerge at about 2–3 months of age (Ellingson & Peters, 1980; Scholle, Zwacka, & Scholle, 2007). The proportion of time infants spent in REM sleep is considerably higher than it is in adults, about 50% (Grigg-Damberger et al., 2007).
Sleep electroencephalography and brain maturation: developmental trajectories and the relation with cognitive functioning
2020, Sleep MedicineCitation Excerpt :Sleep spindle features exhibit an age-dependent pattern [91], and their developmental modifications are presumably related to the maturation of thalamocortical structures [82]. A “rudimentary” spindle activity has been observed in the first two weeks postterm [24,92–94], whereas mature spindles appeared between six weeks and three months, with an increase in most spindle parameters [93,95]. The period between two and three months corresponds to the emergence of a peak in the sigma frequency range [24].
Sleep-wake cycle of the healthy term newborn infant in the immediate postnatal period
2016, Clinical NeurophysiologyCitation Excerpt :Several studies have examined the SWC of healthy term infants during the first weeks/months of birth (Parmelee and Stern, 1972; Ellingson and Peters, 1980; Peirano et al., 1993; Ficca et al., 2000). The only EEG studies that have described the composition of the SWC of healthy term newborn infants, within the first days after birth, were based on relatively small cohorts with EEG recorded between day 1 and 10 after birth (Ellingson and Peters, 1980; Curzi-Dascalova et al., 1988; Scher et al., 2002). The SWC of healthy term infants, within the first hours after birth, has not yet been fully investigated making the interpretation of “abnormalities” in the SWC during this time period difficult.
Do aluminum vaccine adjuvants contribute to the rising prevalence of autism?
2011, Journal of Inorganic BiochemistryCitation Excerpt :In addition, the correlation between ASD prevalence and Al adjuvant exposure appears to be the highest at 3–4 months of age. Of note, these periods (i.e., first 4 postnatal months) coincide with several critical stages of human brain development and biobehavioural transitions that are known to be impaired in autism (i.e., onset of synaptogenesis, maximal growth velocity of the hippocampus [3], onset of amygdala maturation [81] and development of brain-wave and sleeping patterns [82,83]). Clearly, we cannot draw definite conclusions regarding the link between Al adjuvants and autism based on an ecological study such as the present one and hence the validity of our results remains to be confirmed.
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Supported by NIH Research Grant No. HD-09338 from the National Institute of Child Health and Human Development.
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Present address: Boys Town Institute for Communication Disorders in Children, 555 North 30th Street, Omaha, Nebr. 68131, U.S.A.