Factors affecting use of the Doppler-determined time from flow onset to maximal pulmonary artery velocity for measurement of pulmonary artery pressure in children☆
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Cited by (45)
Cardiopulmonary Adaptation During First Day of Life in Human Neonates
2018, Journal of PediatricsCitation Excerpt :In the presence of an open ductus, the latter change may relate to the exposure of the right ventricle to elevated SVR which, despite a decreasing PVR, reflects a marked difference from its intrauterine state, where the placenta was a major contributor to low afterload. Another concern with the use of PAAT in newborns is its strong negative correlation with heart rate,20 which can impact its clinical utility, particularly at rates greater than 100 beats/min.21 Previous validation studies for PAAT as a marker for PVR were conducted in adults and older children10,22 Indexing PAAT to cardiac cycle duration resolved the confounding effect of heart rate in this study; however, PAATi needs further validation with a gold standard, invasive method.
Noninvasive pulmonary arterial pressure estimation using a logistic-based systolic model
2018, Computers in Biology and MedicineCitation Excerpt :As our preliminary suggestion for the future clinical study, due to the greater flexibility of the proposed PAP equation to more accurately estimate the catheterized PAP, i.e. for the middle PAcT values and, simultaneously, for the small and large of the PAcT values, having the actual clinical echocardiography measurement datasets evenly distributed over the very large range of the PAcT domain may be necessary to unleash its statistical significance—for example, having 5–7 measurements in each of PAcT <50 msec, 50–70 msec, 70–90 msec, 90–110 msec, 110–130 msec, 130–150 msec, and >150 msec. Additionally, if possible, a subsequent clinical study should better be designed to accommodate possible underlying confounding factors that influence either PAcT or AAcT by the presence of a ventricular septal defect or pulmonary arterial stenosis, the reduction of afterload [21], heavy exercise [22], vascular resistance [23], heart rate [24], and cardiac disease, including myocardial infarction [25] and cardiomyopathy [26]. Therefore, with stratification on the factors, it may thus be necessary to have a certain range of sample sizes for each interval of PAcT over the very large range of the PAcT domain.
Pulmonary Artery Acceleration Time Provides a Reliable Estimate of Invasive Pulmonary Hemodynamics in Children
2016, Journal of the American Society of EchocardiographyCitation Excerpt :PAAT represents the interactive changes in ventricular mechanical performance, pulmonary vascular load, and compliance in children. Studies in children have previously evaluated PAAT as an echocardiographic measure of PAP, but with inconsistent results and lack of obvious reliable “cutoff” values to establish sensitive interrelation between DE-derived PAAT and RHC-derived pulmonary hemodynamics.2,11,15-18 Kosturakis et al.16 demonstrated PAAT as a discriminator between patients with elevated PAP and normal PAP, with PAAT showing a moderate inverse correlation with elevated PAP (r = −0.62).
Superiority of 10% Air-10% Blood-Saline Mixture for Measuring the Velocity of Tricuspid Regurgitation in Patients with Severe Emphysema
2003, Journal of the American Society of EchocardiographyThe usefulness of a 10% air-10% blood-80% saline mixture for contrast echocardiography: Doppler measurement of pulmonary artery systolic pressure
2002, Journal of the American College of Cardiology40 MHz Doppler characterization of umbilical and dorsal aortic blood flow in the early mouse embryo
2000, Ultrasound in Medicine and Biology
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This work was supported in part by grants HL00718, HL28391, HL11307, and HL07063 from the National Heart, Lung, and Blood Institute, Bethesda, Maryland.