Responses

Download PDFPDF
Size for gestational age at birth: impact on risk for sudden infant death and other causes of death, USA 2002
Compose Response

Plain text

  • No HTML tags allowed.
  • Web page addresses and e-mail addresses turn into links automatically.
  • Lines and paragraphs break automatically.
Author Information
First or given name, e.g. 'Peter'.
Your last, or family, name, e.g. 'MacMoody'.
Your email address, e.g. higgs-boson@gmail.com
Your role and/or occupation, e.g. 'Orthopedic Surgeon'.
Your organization or institution (if applicable), e.g. 'Royal Free Hospital'.
Statement of Competing Interests

PLEASE NOTE:

  • Responses are moderated before posting and publication is at the absolute discretion of BMJ, however they are not peer-reviewed
  • Once published, you will not have the right to remove or edit your response. Removal or editing of responses is at BMJ's absolute discretion
  • If patients could recognise themselves, or anyone else could recognise a patient from your description, please obtain the patient's written consent to publication and send them to the editorial office before submitting your response [Patient consent forms]
  • By submitting this response you are agreeing to our full [Response terms and requirements]

Vertical Tabs

Other responses

Jump to comment:

  • Published on:
    This may be explained by DHEA Levels

    It is my hypothesis that all tissues rely on DHEA for optimal function. Low DHEA has been found in preterm infants (Acta Paediatr. 2007 Nov;96(11):1600-5). I suggest this may be due to reduced availability of maternal DHEA as the infant does not start producing its own DHEA until birth.

    It is also my hypothesis that SIDS results from excessively low DHEA during sleep which results in lack of support of brainst...

    Show More
    Conflict of Interest:
    None declared.