Nitric oxide and lung surfactant
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Cited by (43)
The impact of staphylococcal mastitis on the level of milk IL-6, lysozyme and nitric oxide
2010, Comparative Immunology, Microbiology and Infectious DiseasesCitation Excerpt :The significantly increased production of NO in the clinically mastitic cows could be intimately correlated with the production of staphylococcal enterotoxin C [13]. The increased production of NO and IL-6 which are produced during clinical mastitis under the influence of staphylococcal enterotoxin β [13] and [42] causes down regulation of CYP450 which consequently leads to oxidative damage of the mammary gland in S. aureus-induced mastitis [13,48–51]. Worldwide, a lot of effort has been focused on how to minimize the effects of subclinical and chronic mastitis, for example by preventing intramammary infection using vaccines, or to eliminate ongoing infections by treatment with antibiotics, or to stimulate the immune system of an animal with non-specific immunomodulators.
Effect of antioxidants on apoptosis and cytokine release in fetal rat Type II pneumocytes exposed to hyperoxia and nitric oxide
2004, CytokineCitation Excerpt :So, while NO may be a critical intermediary in the production of oxidant tissue damage [18], NO-dependent protective effects have been observed in processes where oxidant injury has been proposed to play an etiological role [20–22]. A probable explanation for these differing results is that NO is a free radical with both AO and pro-oxidant properties—a molecular chameleon [23]. The dose and duration of exposure to exogenous NO, in the presence or absence of hyperoxia, may determine its beneficial or detrimental role.
Nitric oxide in critical respiratory failure of very low birth weight infants
2004, Paediatric Respiratory ReviewsPhysiology of Nitric Oxide in the Developing Lung
2003, Fetal and Neonatal Physiology: Third EditionNO donors and NO delivery methods for controlling biofilms in chronic lung infections
2021, Applied Microbiology and Biotechnology