Original articleThe relationship of erythrocyte glucose-6-phosphate dehydrogenase deficiency to hyperbilirubinemia in Negro premature infants+
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Increased incidence of severe hyperbilirubinemia among newborn Chinese infants with G-6-PD deficiency
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Cited by (23)
The Preterm Infant. A High-Risk Situation for Neonatal Hyperbilirubinemia Due to Glucose-6-Phosphate Dehydrogenase Deficiency
2016, Clinics in PerinatologyCitation Excerpt :The high-risk nature of preterm, G6PD-deficient newborns for significant hyperbilirubinemia was apparent already in the pre-phototherapy era. Eshaghpour and colleagues26 identified 10 of 87 (11.5%) African American infants admitted to a premature infant nursery in Philadelphia, PA to be G6PD-deficient. Six (60%) of these developed TB levels greater than 20 mg/dL, and exchange transfusion was performed in 5 (50%).
Erythrocyte Disorders in Infancy
2005, Avery's Diseases of the NewbornErythrocyte enzyme disorders in children
1980, Pediatric Clinics of North AmericaUn déficit en glucose 6 phosphate déshydrogénase peut-il à lui seul expliquer un ictère néo-natal ?
1978, Revue Francaise de Transfusion et Immuno-hematologieHemolysis in the newborn infant resulting from deficiencies of red blood cell enzymes: Diagnosis and management
1974, The Journal of PediatricsCongenital hemolytic anemias and red cell enzyme deficiencies
1973, Current Problems in Pediatrics
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Supported in part by United State Public Health Service Grants AM 02231 and HO 01919.
Presented in part at the 76th Meeting of the American Pediatric Society, April, 1966.
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Address, Hospital of the University of Pennsylvania, 3600 Spruce St., Philadelphia, Pa. 19104.
Copyright © 1967 Published by Mosby, Inc.