Lactate in cord blood and its relationship to pH and catecholamines in spontaneous vaginal deliveries
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Venous blood gas parameters in healthy Mediterranean buffalo calves in the first 72 hours of life
2020, TheriogenologyCitation Excerpt :This increase could be induced by the mechanism of parturition. During parturition, an increase in catecholamine secretion was found [43]. These hormones seemed responsible for the glucose increase directly by improving the glycogenolysis via the glucagon release, and indirectly inhibiting insulin secretion [44,45], with glucose availability of both aerobic and anaerobic glycolysis.
Placental distribution of endogenous and exogenous substances: A pilot study utilizing cryo-sampled specimen off delivery room
2020, PlacentaCitation Excerpt :However, higher levels of Cr, Fe, and PCBs were observed in this study [35–37], probably due to the differences in exposure level, blood content, potential contamination during sampling, and sample size. NE in cord blood measured here was about 10 times lower compared with previous study while DA was higher [38]. Monoamines were < LOD in all placenta samples, probably because of the high monoamine oxidase activity in placenta [39].
Lactate in cord blood and its relation to fetal gluconeogenesis in at term deliveries
2015, Early Human DevelopmentCitation Excerpt :Experimental animal data have suggested that lactate concentration is better than pH in predicting hypoxic process and neonatal morbidity [6]. In the human fetus, increased lactate and glucose levels can be anticipated when hypoxia and stress are present and is likely to be a function of both anaerobic metabolism [7–9] and catecholamine-mediated glycogenolysis/glycolysis [10–12]., Interpreting cord blood lactate and glucose levels is therefore complex [13].
Amniotic fluid and blood lactate concentrations in mares and foals in the early postpartum period
2012, TheriogenologyCitation Excerpt :The mean blood lactate concentration of the foals decreased during the first 24 h of life. The high concentration measured at birth, and also reported in human newborns [20,41], could be due to the release of cortisol and catecholamine [42–44] or to physiological hypoxia during the birth process. This is supported by the very low arterial lactate concentration of the equine fetus (1.0 ± 0.20 mmol/L) from mid to late gestation [10].
Venous blood lactate evaluation in equine neonatal intensive care
2010, Theriogenology