1 | 25 | 19 days | PHH | C | Bilateral IVH, left venous infarction, secondary PHH | |
| | | | P | Perfusion not detected in WM; well characterised in GM | |
2 | 34 | 3 days | HIE injury | C | Bilateral IVH, bilateral venous infarction, cortical highlighting, oedema | |
| | | | P | Extensive perfusion voids in WM | |
3 | 35 | 3 days | Sialoblastoma | C | Large soft tissue mass without intracranial extension | |
| | | | P | Small perfusion voids in WM and some in GM | |
4 | 37 | 14 days | Convulsions | C | Normal | |
| | | | P | Small perfusion voids in WM; perfusion well characterised in GM | Fig 2D |
5 | 38 | 7 months | Meningococcal meningitis | C | Normal | |
| | | | P | Perfusion well characterised in GM and WM | |
6 | 39 | 8 days | Convulsions | C | Extensive cortical highlighting and WM oedema | |
| | | | P | Perfusion well characterised in GM and WM | |
7 | 39 | 23 days | Convulsions | C | Normal | |
| | | | P | Extensive perfusion voids in WM; perfusion well characterised in GM | |
8 | 39 | 18 months | Di George syndrome | C | Severe hydrocephalus | Fig 1A,C |
| | | | P | Perfusion well characterised in GM and WM; voids due to enlarged ventricles hypoxic ischaemic encephalopathy | Fig 1B,D |
9 | 40 | 2 days | Axial hypotonia | C | Normal | |
| | | | P | Minimal perfusion voids in WM | |
10 | 40 | 4 days | Disraphysm | C | Several punctate lesions in WM | |
| | | | P | Minimal perfusion voids in WM | |
11 | 40 | 5 months | Convulsions | C | Slightly prominent lateral ventricles-otherwise normal | |
| | | | P | Perfusion well characterised in GM and WM | |
12 | 41 | 13 days | HIE injury | C | Extensive and generalised WM oedema | Fig 2A |
| | | | P | Perfusion voids in frontal WM and focally in cerebral cortex | Fig 2B |
| 41 | 69 days | HIE injury | C | Cystic change in posterior parieto-occipital region; atrophic changes | Fig 2C |
| | | | P | Not available | |