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Intrauterine growth retardation (IUGR) is an important cause of neonatal mortality, morbidity and poor neurological outcome.1 Hypoglycaemia as a consequence of IUGR is a major risk factor for neurodevelopmental impairment. The factors that predispose these patients to hypoglycaemia include failure of counter-regulation, immaturity of the enzyme systems regulating glycogenolysis, gluconeogenesis, ketogenesis, reduced adipose tissue stores, hyperinsulinism or increased sensitivity to insulin.2–4 Hypoglycaemia in patients with IUGR is usually thought to be transient, lasting for a few days. However, the precise duration …
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