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The Scottish perinatal neuropathology study: clinicopathological correlation in early neonatal deaths
J C Becher, J E Bell, J W Keeling, N McIntosh, and B Wyatt
Arch. Dis. Child. Fetal Neonatal Ed. 2004 89: F399-407.

Publisher Correction

PLease note there is an error in Table 4 (second row, last column). The correct version of table is shown below:


Table 4  Histological evidence of brain damage, including putative prenatal damage, in 59 neonates aged 3 days or less

Pathological feature Encephalopathic
No encephalopathy
No encephalopathy
BA group (n = 10; 17%)
BA group (n = 36; 61%)
NoBA group (n = 13; 22%)
Mature
(n = 8)		 Preterm
(n = 2)		 Mature
(n = 13)		 Preterm
(n = 23)		 Mature
(n = 2)		 Preterm
(n = 11)
Neuronal eosinophilia 8 (100) 1 5 (38) 4 (17) 0 4 (36)
Neuronal karyorrhexis 8 (100) 1 1 (8) 4 (17) 0 0
Grey matter infarcts 0 0 0 0 0 0
White matter gliosis* 7 (88) 1 4 (31) 9 (39) 1 0
Grey matter gliosis* 5 (63) 1 0 1 (4) 0 0
Microglial upregulation 6 (75) 1 2 (15) 9 (39) 0 1 (9)
Macrophages* 7 (88) 1 1 (8) 8 (35) 0 0
Fresh haemorrhage 4 (50) 0 6 (46) 11 (48) 0 5 (45)
Haemosiderin deposits 0 0 0 0 0 0
Mineral deposits 1 (13) 0 1 (8) 6 (26) 1 1 (9)
*Estimated prenatal brain damage 8 (100) 1 5 (38) 12 (52) 1 0
Values in parentheses are percentages.
Mature, >=37 weeks; preterm, 24?36 weeks.


 

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